Astaxanthin, a carotenoid with potential in human health and Nutritional value and Application
Astaxanthin
a red-orange carotenoid pigment, is a powerful biological antioxidant that occurs naturally in a wide variety of living organisms. The potent antioxidant property of 1 has been implicated in its various biological activities demonstrated in both experimental animals and clinical studies. Compound 1 has considerable potential and promising applications in human health and nutrition. In this review, the recent scientific literature (from 2002 to 2005) is covered on the most significant activities of 1, including its antioxidative and anti-inflammatory properties, its effects on cancer, diabetes, the immune system, and ocular health, and other related aspects. We also discuss the green microalga Haematococcus pluvialis, the richest source of natural 1, and its utilization in the promotion of human health, including the antihypertensive and neuroprotective potentials of 1, emphasizing our experimental data on the effects of dietary astaxanthin on blood pressure, stroke, and vascular dementia in animal models, is described.
demonstrated that astaxanthin inhibited bodyweight gain in a diet which contained ten times the amount of fat (without changing the energy intake) when compared to the controlled group (Figure 2). The sixty-day study revealed several significant improvements such as total body weight (15% less), adipose tissue (34% less), liver triglycerides (58% less), plasma triglyceride, liver weight and total cholesterol. In addition, it appears that weight-gain and adipose tissue gains were suppressed in a dose dependent manner. Based on the respiratory quotient, the inhibition of weight-gain may be linked to increased thermogenesis and fat utilization.
more:demonstrated that astaxanthin inhibited bodyweight gain in a diet which contained ten times the amount of fat (without changing the energy intake) when compared to the controlled group (Figure 2). The sixty-day study revealed several significant improvements such as total body weight (15% less), adipose tissue (34% less), liver triglycerides (58% less), plasma triglyceride, liver weight and total cholesterol. In addition, it appears that weight-gain and adipose tissue gains were suppressed in a dose dependent manner. Based on the respiratory quotient, the inhibition of weight-gain may be linked to increased thermogenesis and fat utilization.
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